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Thread: more good canna news-anti propoganda page

  1. #11

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    Quote Originally Posted by tokers_united View Post
    So COME ON, these scientific study posts are BRILLIANT, keep them coming and above all keep the credibility and accuracy. Don't post here say that isn't backed up by sources. We are better than that and soon the world will realise credible source over paranoia and propoganda and then we will have a voice.
    This one.

    Don't politicize it, don't exaggerate, and don't lie.

    And, for christ's sake, don't think that exposing mental disorders through excessive cannabis use is the first step to treating them.
    "Certain
    Kinds of people that we like to know, yeah
    And others gotta go
    If you wait 'till you're ready, then you'll never make an amends"

  2. #12
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    Quote Originally Posted by yavana View Post
    This one.

    Don't politicize it, don't exaggerate, and don't lie.

    And, for christ's sake, don't think that exposing mental disorders through excessive cannabis use is the first step to treating them.
    lol that i admit that was a stoner 'give me back my weed' line
    but u cant approach it in any other way now...

    if u want to do this properly join LCA

  3. #13

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    Quote Originally Posted by Wellstoned View Post
    We're still battling the notions created by the original Reefer Madness propaganda I think
    that film was fucking hilarious .probably one of my favourite weed films.
    i got sunshine in a bag and im useless

  4. #14

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    This is a very interesting read >>

    Therapeutic Uses of Cannabis

    • Extracts of the cannabis plant (cannabinoids) are thought to act primarily on CB1 and CB2 receptors.1
    • CB1 receptors are found mainly in the central nervous system, but also in dorsal root ganglia, sympathetic ganglia, adrenal gland, heart, lung, reproductive tissues, urinary bladder, gastrointestinal tissues. They are found on pain pathways in the brain and spinal cord, as well as terminals of peripheral nervous system primary afferent neurons.
    • An entire endocannabinoid system has now been identified modulating brain regions with different functions in responses to aversive stimuli.2
    • CB2 receptors are principally located in immune cells.
    • Cannabinoid receptors play an important role in a variety of physiological processes including metabolic regulation, craving, pain, anxiety, bone growth, and immune function.
    • Naturally-occurring chemicals (endocannabinoids) have been identified which are thought to act as 'retrograde synaptic messengers', being released by post-synaptic neurones and travelling backwards to suppress pre-synaptic neurotransmitter release. Endocannabinoids regulate the release of other neurotransmitters that may have even opposing functions, such as GABA and glutamate.2
    • The results of stimulating the endocannabinoid system are poorly understood. The most widely known is the potential for producing well-being (the 'bliss' reaction) and in reducing pain due to muscle spasm, these effects being explored for their therapeutic potential. However, stimulation can also produce anxiety and panic. The variation in effect may be dependent on the emotional state of the subject at the time.2
    • Current research is exploring the use of endogenous arachidonic acid metabolites related to endocannabinoids, in the treatment of cancer. They have been found to have inhibitory effects on tumour growth, angiogenesis, migration and metastasis. Remarkably, these effects seem isolated to cancer cells and there are no inhibitory effect on the proliferation of normal cells.3
    • Other uses of cannabis and related compounds include hypnotics, analgesics, antiemetics, antiasthmatics, antihypertensives, immunomodulatory drugs, antiphlogistics, neuroprotective agents, antiepileptics, agents influencing glaucoma, spasticity, alcohol withdrawal, hepatic fibrosis, bone growth, and atherosclerosis.4

    Sativex5
    The only cannabis-related preparation currently available for legal medical use is Sativex.
    It was granted a Notice of Compliance with Conditions in Canada (the equivalent of a UK Medicines Act license) in April 2005.
    It remains an unlicensed drug in the UK and in the rest of Europe. The last official comment from the Medicines Healthcare Regulatory Agency (MHRA) was a statement in December 2007. This clarified that an application for a marketing authorisation for Sativex Oromucosal Spray had been submitted by the manufacturers in August 2006. Before that the product been supplied on a named patient basis in the UK. As of December 2007 1200 patients had been prescribed it on this basis. The report stated that whilst sufficient information had been submitted concerning the pharmacodynamics of the drug, further evidence about the efficacy and safety of Sativex would be required before a license could be considered, should the manufacturer choose to submit it.
    Sativex is a buccal spray containing the active ingredients delta-9-tetrahydrocannabinol 27 mg/ml and cannabidiol 25 mg/ml, which are both extracts of the hemp plant Cannabis sativa Linne.
    Sublingual tablets and an inhaled preparation were being investigated but there is no recent information concerning these routes.6

    Indications5


    Contraindications

    • Known or allergy to cannabinoids or other constituents of Sativex (propylene glycol, ethanol or peppermint oil)6
    • Patients with significant hepatic disease (due to the ethanol content), renal dysfunction (cannabinoids are excreted via the kidney)6
    • Serious cardiovascular disease, e.g. IHD, significant arrhythmias, unstable hypertension, severe cardiac failure (sympathoexcitation, bradycardia and cardiovascular depression in animal experiments)11
    • A history of schizophrenia or other psychotic disorder
    • History of substance abuse including alcohol abuse - Sativex has addictive potential and should be avoided6
    • Reproductive issues:
      • Evidence that even mild to moderate cannabinoid use in pregnancy is associated with symptoms of ADHD (inattention, impulsivity), increased externalising behavior, decreased general cognitive functioning, and deficits in learning and memory tasks.12
      • Women of reproductive age should use reliable contraception
      • Spermatogenesis can be affected13
      • Male and female patients should maintain reliable contraception for the duration of therapy and for three months after14
    • Lactation - degree with which cannabinoids are excreted in breast milk is unknown,6 but it is thought likely that the risk is significant. Sativex is therefore contraindicated in lactating mothers.5
    • Safety and efficacy of Sativex not yet been established in patients under the age of 18.14

    Warnings and special precautions5

    • Warn patients they may get 'intoxication-type reactions' - e.g. sudden changes in mood, decreased cognitive performance and memory, alteration in perception of reality and time, and lack of inhibition. These effects are dose-related.
    • Patients should be warned not to drive or do any activity which involves judgement or co-ordination whilst taking Sativex.
    • Use with caution in patients with pre-existing heart disease, arrhythmias, cardiac failure, poorly controlled hypertension, bradycardia, tachycardia and postural hypotension.
    • Epilepsy - there is evidence that seizure threshold is related to activity at CB1 and CB2 receptor sites.
    • There is limited data on use in the elderly, so patients should be carefully monitored.

    Dosage and administration6

    • Patients should start with one spray every four hours up to a maximum of four sprays a day, and then self-titrate according to response.
    • Warn the patient that it can take a week or more to find the right dose and to watch for symptoms of intoxication.
    • Pre-marketing trials found that the average dose used was five sprays a day. There is limited evidence with doses higher than twelve sprays daily, although some patients may require this.
    • The patient should spray directly below the tongue or towards the inside of the cheek, varying the site at each dose. Avoid spraying on inflamed areas. The spray should never be aimed at the pharynx as this can cause irritation.

    Adverse effects6

    • The commonest adverse effects during pre-marketing trials were headache, impaired balance, depressed mood and irritation at the site of administration.
    • Syncopal attacks relating to postural hypotension have been identified.
    • One case of a causal relationship between Sativex and suicidal ideation could not be ruled out, but the incidence of depression was consistent with that observed in populations of multiple sclerosis patients followed for a prolonged period of time.
    • Hallucinations, episodes of paranoia and other psychotic symptoms have also been reported.

    Monitoring6

    No routine laboratory monitoring is required other than that necessary to monitor the patient's condition and any concomitant medication. The patient should however be assessed regularly to see whether continued administration of Sativex is appropriate, due to its addictive potential.6

    Interactions5

    • Sativex can potentiate the action of opioids, particularly fentanyl and alfentanil, and amitriptyline.
    • Cannabinoids are also known to have an affect on drugs which rely on protein-binding transport systems, such as verapamil, so post-marketing trials are likely to identify many more interactions in the future.6,15

    Prescribing an unlicensed drug16,17

    A drug which is not yet licensed for UK use can be prescribed on a named patient basis, but be aware of the following issues:
    • The doctor who signs the prescription takes full responsibility for the outcome and cannot cite the pharmaceutical company as co-defendant in any civil legal action arising from a serious untoward event.
    • Make sure that both you and the patient are fully conversant with the contra-indications, special precautions, and adverse reactions.
    • Record the issues you discussed with the patient.
    • Monitor the patient regularly.


    Document references
    1. Mackie K; Cannabinoid receptors as therapeutic targets.; Annu Rev Pharmacol Toxicol. 2006;46:101-22. [abstract]
    2. Moreira FA, Lutz B; The endocannabinoid system: emotion, learning and addiction. Addict Biol. 2008 Jun;13(2):196-212. Epub 2008 Apr 16. [abstract]
    3. Flygare J, Sander B; The endocannabinoid system in cancer-potential therapeutic target? Semin Cancer Biol. 2008 Jun;18(3):176-89. Epub 2007 Dec 14. [abstract]
    4. Svizenska I, Dubovy P, Sulcova A; Cannabinoid receptors 1 and 2 (CB1 and CB2), their distribution, ligands and functional involvement in nervous system structures - A short review. Pharmacol Biochem Behav. 2008 May 25. [abstract]
    5. Public Information Report on Sativex Oromucosal Spray; MHRA December 2007.
    6. No authors listed; Cannabis-based medicines--GW pharmaceuticals: high CBD, high THC, medicinal cannabis--GW pharmaceuticals, THC:CBD.; Drugs R D. 2003;4(5):306-9. [abstract]
    7. Berman JS, Symonds C, Birch R; Efficacy of two cannabis based medicinal extracts for relief of central neuropathic pain from brachial plexus avulsion: results of a randomised controlled trial.; Pain. 2004 Dec;112(3):299-306. [abstract]
    8. RxPG News
    9. Blake DR, Robson P, Ho M, et al; Preliminary assessment of the efficacy, tolerability and safety of a cannabis-based medicine (Sativex) in the treatment of pain caused by rheumatoid arthritis.; Rheumatology (Oxford). 2006 Jan;45(1):50-2. Epub 2005 Nov 9. [abstract]
    10. Niederhoffer N, Szabo B; Cannabinoids cause central sympathoexcitation and bradycardia in rabbits.; J Pharmacol Exp Ther. 2000 Aug;294(2):707-13. [abstract]
    11. Niederhoffer N, Schmid K, Szabo B; The peripheral sympathetic nervous system is the major target of cannabinoids in eliciting cardiovascular depression.; Naunyn Schmiedebergs Arch Pharmacol. 2003 May;367(5):434-43. Epub 2003 Apr [abstract]
    12. Huizink AC, Mulder EJ; Maternal smoking, drinking or cannabis use during pregnancy and neurobehavioral and cognitive functioning in human offspring. Neurosci Biobehav Rev. 2006;30(1):24-41. Epub 2005 Aug 10. [abstract]
    13. Nahas GG, Frick HC, Lattimer JK, et al; Pharmacokinetics of THC in brain and testis, male gametotoxicity and premature apoptosis of spermatozoa.; Hum Psychopharmacol. 2002 Mar;17(2):103-13. [abstract]
    14. Health Canada; Approval of SATIVEX® with Conditions. Fact sheet 2005.
    15. Zhu HJ, Wang JS, Markowitz JS, et al; Characterization of P-glycoprotein inhibition by major cannabinoids from marijuana.; J Pharmacol Exp Ther. 2006 May;317(2):850-7. Epub 2006 Jan 26. [abstract]
    16. British Medical Association; Therapeutic uses of cannabis. 1997
    17. MTRAC; Midlands Therapeutic Review and Advisory Committee Guidance on Unlicensed Prescribing 2005

  5. #15

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    Nice one lk, I found some interesting research too with regards to the governments 'BIG' excuse that cannabis causes schizophrenia/mental illness even though cases have remained constant or even lowered. There have been large studies of generations of people (in other countries), and also of twins that have found that (although no study should ever claim to be conclusive only significant) cannabis is not causal and the mental health problems would have happened anyway, and that enviromental factors play a large part too. Unfortunately the paper does not appear in full but the Familial Predisposition for Psychiatric Disorder study is still documented as:

    Comparison of Subjects Treated for Cannabis-Induced Psychosis and Schizophrenia

    Mikkel Arendt, MScPsych, PhD; Preben B. Mortensen, DrMedSc; Raben Rosenberg, DrMedSc; Carsten B. Pedersen, MSc; Berit L. Waltoft, MSc
    Based on the findings, the researcher says, "cannabis-induced psychosis is probably not a valid diagnosis. It should be considered schizophrenia."

    http://www.theregister.co.uk/2008/11...ychosis_study/
    http://www.reuters.com/article/healt...rpc=22&sp=true
    http://archpsyc.ama-assn.org/cgi/con...269&view=short




  6. #16

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    Default Alcohol Vs Marijuana

    Alcohol Vs Marijuana
    Which is more detrimental to the British public?

    (all information used is as close to government sources as I could possibly find to avoid unauthentic issues raised & questioned)


    Alcohol

    “The Strategy Unit’s analysis last year showed that alcohol-related harm is costing
    around £20bn a year,
    and that some of the harms associated with alcohol are getting
    worse.”
    Tony Blair (statement published 2004 so is with regards to 2003)
    (http://image.guardian.co.uk/sys-file...olstrategy.pdf)
    (Note:This financial figure will have an offset due to the revenue tax & duty charges on imported alcohol of which as of yet I cannot find a valid government figure for.)


    In 2007, the male alcohol-related death rate was 18.1 deaths per 100,000 population, more than twice the rate for females of
    8.7 per 100,000. Males accounted for approximately two-thirds of the total number of deaths. There were 5,732 alcohol-related deaths in men and 2,992 in women. The rate of male deaths has almost doubled from 9.1 per 100,000 population in 1991
    (http://www.statistics.gov.uk/cci/nugget.asp?id=1091)

    The annual cost of alcohol misuse includes:
    • 1.2m violent incidents (around half of all violent crimes);
    • 360,000 incidents of domestic violence (around a third) which are linked to
    alcohol misuse;
    • increased anti social behaviour and fear of crime – 61% of the population perceive
    alcohol-related violence as worsening;
    • expenditure of £95m on specialist alcohol treatment;
    • over 30,000 hospital admissions for alcohol dependence syndrome;
    • up to 22,000 premature deaths per annum;
    • at peak times, up to 70% of all admissions to accident and emergency
    departments;

    • up to 1,000 suicides;
    • up to 17m working days lost through alcohol related absence;
    • between 780,000 and 1.3m children affected by parental alcohol problems; and
    • increased divorce - marriages where there are alcohol problems are twice as likely
    to end in divorce.
    ( http://image.guardian.co.uk/sys-file...olstrategy.pdf)












    Marijuana


    Currently, the number of cannabis users in the UK is estimated at more than three million.
    (http://www.homeoffice.gov.uk/rds/pdfs05/hosb1605.pdf)


    Studies have varied in the methods used to detect psychotic symptoms.
    Only one study has had the statistical power to assess whether
    cannabis use precedes the onset of an illness that meets the full
    diagnostic criteria for schizophrenia.
    The Council concluded that the evidence supports a causal association
    between the use of cannabis, in adolescence, and the later development
    of schizophrenia; although the evidence for this relationship is clearly
    more complicated than when it considered this previously.
    The Council also considered that the evidence supporting a dose-response
    relationship was more persuasive than previously. The Council remains
    uncertain about whether early cannabis use, before the age of 15 years,
    is associated with an additional increased risk. Nevertheless, the
    magnitude of the effect of cannabis use on the subsequent development
    of schizophrenia does not appear to be substantial, in the population as
    a whole
    , with the cannabis preparations used during the late 1990s.
    The peak incidence of schizophrenia in males (aged 20 to 24 years) is
    approximately 15.9 per 100,000 person years, and in females (where
    the peak incidence is at age 25 to 29 years) the peak incidence is 7.5
    per 100,000 person years. Assuming that heavy users of cannabis
    have a two-fold increased risk of developing schizophrenia, based on the
    model discussed earlier, it can be estimated that the increase in
    annual risk:
    • for males (aged 20 to 24 years) would be 1 in 3,100 to 1 in 1,900;
    and
    • for females (aged 25 to 29 years) would be 1 in 9,900 to 1 in 5,300.
    (http://drugs.homeoffice.gov.uk/publi...08?view=Binary)

    (NOTE: although no Government statistics have yet been published on this website that show annual deaths relating to Marijuana consumption alone, a link between cancer and smoking in general is understood to be quite high.
    Also its worth mentioning Gordon Brown made reference to "lethal" strains of Cannabis on British streets on GMTV's morning show around the time of re-classification which contradicts having no figures or statistics to show anyone having died from what is labeled as lethal)

    (NOTE: No government figures could be found on this website detailing the cost or financial burden of Marijuana consumption alone to the British tax payer through policing measures to combat Marijuana neither any figure as to the cost to the NHS, although I understand there is some economical benefit derived from fines issued as a penalty for possession of cannabis)

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  8. #17

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    Default Cannabis houses

    Green really is green, I know we all know it but its good to see them using it for one of the many many other uses it has.

    http://ecoworldly.com/2009/04/14/can...bon-footprint/




  9. #18

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    Good for your skin too, it goes on and on, stop fucking ignoring the god plant governments.
    Love this line:
    "This research shows that we may have something in common with the marijuana plant," said Gerald Weissmann, MD. "Just as THC is believed to protect the marijuana plants from pathogens, our own cannabinoids may be necessary for us to maintain healthy skin and to protect us from pathogens ."
    The anti cancer effects come up a lot gonna have to stop smoking it and eat it instead me thinks.
    http://esciencenews.com/articles/200...uana.good.skin




  10. #19

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    this is a really great thread guys!!

    respect to u dekay for posting the FACTS lol(dont mean much from nu member lol)

    ive been quite ignorant to the whole battle about legalising(through choise) as even 5 years ago the whole movement & us wasnt taken serious, but now we have respected members of socity backing us like doctors, solicitors, even govement workers! its just a matter of time

    from reading through the site the last couple weeks ive seen daily mail mentioned alot as pushing all the lies & 'studies' to scare us all(or well the public)

    i like the ideas u guys have about posting in the comments where u can, as sum1 said we just need to bombard these stories with the facts & refrences to back up what we say! if we do it well people will listen

    makes me wish i had the money to start printing my own small daily "alturnative culture" paper(not mag) or we need to start a daily paper called "World Marijuana News"

    would be hard for even the anti-cannabis fuckers to avoid the FACTS if the was a daily marijuana news paper full of undisputible facts & reports, studies & ofcourse genuine refences lol
    Last edited by Superjoint 420; 21-01-10 at 05:52 AM.

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